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ARBs can inhibit the actions of Ang-II on the AT 1 -receptor, whether it is produced by ACE or another enzyme such as cardiac chymase (4, 24, 31)

At minimum, someone considered the biological plausibility, the potential performance angle, or the risk profile serious enough to act

Environmental stressors further modulated susceptibility: high ammonia levels damaged mucosal barriers and impaired tight junctions 107 , while thermal stress activated the hypothalamicpituitaryadrenal axis, inducing stress-related immunosuppression and weakening respiratory mucosal defense 108

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