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Research across multiple decades has focused on GHK-Cus role in collagen and elastin synthesis, glycosaminoglycan production, wound closure, angiogenesis, and more recently, gene expression modulation where data indicates influence over a remarkably broad range of biological pathways

Taken together, the progressive elucidation and investigation of ALS-related disease-causing genes, the successful construction of disease models through the utilization of technologies such as exon sequencing and gene editing, and the further investigation of the potential molecular regulatory mechanisms between ALS mutant genes and mitochondria will facilitate the opening of a new avenue for the study of targeted therapy for ALS disease

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