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MOTS-c acts as a metabolic coordinator, linking mitochondrial health to nuclear gene expression, a critical communication system that often fails during aging and metabolic disease

This paper investigates the malignant progression of BE to EAC by whole-exome sequencing and shows that the emergence of EAC follows a more rapid pathway that involves genome duplication

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Tzeravini E, Simati S, Anastasiou IA, Dalamaga M, Kokkinos A