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In some cases, neurodevelopmental dysplasia may be a secondary process leading to brain dysfunction ( Endogenous neurogenesis after neonatal HIBD It was recently shown that post-neonatal HIBD can induce NSCs proliferation in neurogenesis sites, with subsequent migration of proliferating progenitor cells to a damaged brain region where they acquire the desired phenotype

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The theoretical mechanism involves reduced stomach acid, but clinical significance remains unclear

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