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The release of Cu into tumor cells enhances reactive oxygen species (ROS) as well as nitric oxide (NO) production, which results in DNA fragmentation, induces pro-apoptotic protein upregulation such as Bax, and enhances caspase-3, caspase-8, and caspase-9 generation that motivates cancer cells apoptosis [68] even in solid tumor hypoxic condition [69]

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Chlorogenic acid inhibits the binding of Nrf2 to its repressor protein Keap1 to activate the Nrf2 antioxidant signaling pathway, thereby preventing APAP-induced hepatotoxicity (Wei et al., 2018)

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