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The mechanisms through which progestins exert their therapeutic effects are the anti-estrogenic effects, anti-vasculogenic action, induction of apoptotic cascade in endometriotic cells, suppression of inflammatory pathways (as production of metalloproteinases), cell proliferation suppression, and also anti-neurogenic effects with inhibition of nerve growth within endometriotic lesions [59, 63, 64]

MDA-T32 cells exhibited RSL3-induced, disproportionately robust rise in intracellular ROS levels in concentration-dependent manner when compared to control HThF cells

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