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Because a perfectly measured dose of degraded peptide delivers nothing

Because AOD-9604 does not suppress appetite mechanisms in the first place, there is no adaptation of hunger-regulating neurons and no compensatory hunger amplification when the peptide is stopped

The discovery that NNMT expression is dramatically elevated in adipose tissue of obese humans and animal models, and that NNMT inhibition or knockdown prevents diet-induced obesity and improves metabolic health, has catalyzed intense research interest in NNMT as a therapeutic target for obesity, metabolic syndrome, and related conditions