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It was demonstrated that in the spinal cord of the SOD1-G93A ALS mouse model, the mitochondrial receptor SQSTM1/p62 is recruited to the mitochondrial surface, where it activates mitophagy, whereas knockdown of the Parkin gene reduces the loss of motor neurons, thereby delaying the progression of disease in SOD1-G93A mice (Palomo et al., 2018)

Tendons in particular are slow to heal, as Cleveland Clinic notes in its overview of why injured tendons recover slowly, which is part of why chronic tendinopathy is a common reason patients ask about regenerative peptides

Acknowledgements The authors thank all past and present members of the Schmitt laboratory for the many contributions over the years, and apologize to authors whose work could not be cited owing to space limitations