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Ilg W, Synofzik M, Brotz D, Burkard S, Giese MA, Schols L

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Elevated iron in the SNpc, reduced glutathione, and widespread oxidation of lipids document the oxidative milieu in post-mortem PD brains, whereas deficits in complex I activity and impaired mitophagy underscore the pivotal role of dysfunctional mitochondria in reactive oxygen species (ROS) overload