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Additional studies have found that loss of forkhead box protein P1 ( Foxp)1 and forkhead box protein P4 (Foxp4) in mice can induce ectopic expression of NPY in airway epithelia, resulting in airway hyperresponsiveness [172]

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Histological analyses on miR-181b overexpressing xenografts revealed a decrease of GLUT1 expression and viceversa for miR-181b depleted tumors [27]

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