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Collectively, these results demonstrate that USP2 deletion, either globally or specifically in TECs, protects against UUO-induced renal fibrosis and ferroptosis, likely through regulating the ubiquitination of TFRC and TXNDC5, and possibly via its own lactylation

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The inflammation is a critical component of the progression and maintenance of the pathophysiology of neuropathic pain [6], in which the Wallerian degeneration of axons and myelin sheaths occurring after nerve injury is accompanied by the secretion of inflammatory cytokines, chemokines, and oxidative mediators, the recruitment of macrophages and other immune cells, and the activation of resident astrocytes and microglial cells [7]