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The hypoxic microenvironment of RA joints can cause reactive oxygen species (ROS) accumulation and mitochondrial damage, which not only affect the metabolic processes of immune cells and pathological changes in fibroblastic synovial cells but also upregulate the expression of several inflammatory pathways, ultimately promoting inflammation

2024;S2090-1232(24):001814

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Additionally, it possesses antioxidant characteristics, eliminating ROS and lowering oxidative stresses, which act as a protective mechanism in chronic disease development (Prez-Glvez et al