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For BV-2 cells pretreated with -EC (1, 10, 100, and 1000 M), a significant reduction in LPS-induced NO production was observed specifically in cells treated with 100 M -EC compared to the LPS-stimulated control cells (Fig

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The compound appears to maintain NO homeostasis under conditions of both excess and deficiency upregulating eNOS-derived NO in ischemic models while attenuating iNOS-driven overproduction in inflammatory contexts (Sikiric et al., 2014, Current Pharmaceutical Design 2014