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By influencing NNMT-associated pathways, it serves as a valuable tool in in vitro studies examining metabolic regulation, mitochondrial function, and potential applications in obesity and age-related research contexts

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ERS promotes Mcl-1 downregulation through the PERK/ATF4/CHOP axis while activating the calpain/caspase-12 pathway, forming synergistic crosstalk with mitochondrial apoptosis ( via p21 upregulation and CDK suppression) exacerbate genomic instability and proliferation inhibition (Zhang H