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From an experimental design perspective, the key hypothesis is not that the peptides duplicate each others effects, but rather that they might support different rate-limiting steps in repair, such as cell recruitment to the injury site, fibroblast function, extracellular matrix organization, angiogenesis, and the transition from inflammatory signaling to remodeling

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Individual responses, safety profiles and pharmacokinetic parameters can vary significantly between species and between in vitro and in vivo models

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