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For instance, upon stimulation of myeloid cells, NADPH is significantly depleted due to consumption by inflammatory and anti-inflammatory pathways [25]

For example, TLR2/TLR4 antagonists can reduce overactive inflammation in infected wounds, while topical TLR agonists can enhance pathogen recognition and immune activation in immunocompromised conditions [36]

It serves as an essential cofactor for numerous enzymatic reactions, including the function of superoxide dismutase (SOD), a critical endogenous antioxidant enzyme that neutralizes harmful free radicals before they can damage retinal cells

Regarding ECD, especially in the context of CVDs, ECM remodeling is an important area of interest due to its relationship with alterations in the normal function of ECs