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Current literature reveals wide variation in route of administration, dose intensity, treatment duration, and frequency, even for the same peptide

demonstrated that -synuclein is capable of inhibiting amyloid-beta efflux, suggesting that LRP-1 might be involved in -synuclein efflux as well [131]

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As anticipated, the data in Figure 25 demonstrate that both D-Nle-Cys-Ile-NH-(CH 2 ) 5 -CONH 2 and D-Nle-Tyr-He-NH-(CH 2 )s-CONH 2 , which bind HGF with high affinity (Figure 23) and effectively block its dimerization (Figure 24) were able to block Met auto-phosphorylation