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Currently, targeted neuroprotective drugs acting on ferroptosis pathways are under progressive development, with iron chelators, antioxidants, and free radical scavengers being extensively investigated to achieve neuronal protection through modulation of ferroptosis processes (224)

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-LA, a naturally occurring dithiol with antioxidant and anti-inflammatory function, can decrease the interaction between Nrf2 and Keap1, inhibit the pro-inflammatory cytokine IL-8 production and minimize the infection via the Nrf2/HO-1 pathway in the AGS cells (161)