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Monitoring reduced GSH and its ratio with oxidized GSSG may be used to evaluate the redox and detoxification status of cells and tissues in relation to the protective role of glutathione against oxidative and free-radical-mediated cell injury

The primary mechanism documented in published literature is the induction of telomerase catalytic subunit (hTERT) expression in human somatic cells, resulting in measurable telomerase enzyme activity and telomere elongation

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Clinically, this results in increased blood pressure and heart rate due to unopposed sympathetic stimulation