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Thus while a build up of cytosolic GSH occurs, the inability to transport this into mitochondria is caused by physicochemical alterations to the inner mitochondrial membrane caused by long term alcohol exposure

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Figure 3 Discussion Patients with moderate to severe TBI frequently experience persistent neurocognitive deficits, including short-term memory loss, executive dysfunction, and slowed cognitive processingsymptoms for which there are currently no FDA-approved treatments

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