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Further exploration of the pathways through which ROS influences mitophagy to alter the progression of ischemic cardiomyopathy has shown that inhibition of Parkin-mediated mitophagy leads to excessive mitochondrial ROS accumulation, promoting myocardial injury and heart failure [85]

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General method for carbonheteroatom cross-coupling reactions via semiheterogeneous red-light metallaphotocatalysis

Among these, ferroptosis and mitochondrial damage have emerged as new areas of focus in recent years and are considered potential new therapeutic targets for DCM treatment