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Recently, evidence has emerged linking the absence of the osteocalcin gene with metabolic dysfunctions such as hyperglycemia, glucose intolerance, increased fat mass and serum triglyceride levels, and decreased production of cells, insulin secretion, and adiponectin expression

By modulating key pathways such as neuroinflammation, oxidative stress, and neurotransmitter regulation, these interventions offer promising complementary or alternative therapeutic strategies for AD management

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