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Concluding remarks Although epidemiological evidence suggests that G6PD-d worsens atherosclerosis, the mechanisms by which this occurs are unknown

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Mitochondria-lysosome contact site dynamics and misregulation in neurodegenerative diseases

117 Another example is that the AMPs, human -defensins, can specifically bind to fibronectin (FN) to block the adhesion and transfer of integrin 51 mediating endothelial cells to fibronectin, and block the proliferation and apoptosis of endothelial cells mediated by vascular endothelial growth factor (VEGF), that is, human -defensins inhibit tumor angiogenesis in many ways, thereby exerting its anti-cancer effect