Available at: Summary Keywords dipeptidyl peptidase 1, cathepsin C, elastase, neutrophil, inflammation, pathophysiology, treatment strategy, respiratory disease Citation Chalmers JD, Kettritz R and Korkmaz B (2023) Dipeptidyl peptidase 1 inhibition as a potential therapeutic approach in neutrophil-mediated inflammatory disease
Inhibition of LDH-A by oxamate enhances the efficacy of Anti-PD-1 treatment in an NSCLC humanized mouse model
Quiescent T cells predominantly depend on oxidative phosphorylation and fatty acid catabolism for their energy needs ( Upon activation, T cells rapidly enhance their glycolytic machinery, facilitated by a diverse ensemble of enzymes and proteins
Despite its spread throughout the world, the mechanisms that determine its onset are still to be defined