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Cardiovascular risk factors and molecular routes underlying endothelial dysfunction: novel opportunities for primary prevention

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Demir-Dora D, Ozsoy U, Yildirum Y, Yilmaz O, Aytac P, Yilmaz B, et al

Under physiological conditions, InsRec metabolic function is regulated by negative feedback through tyrosine phosphatases that counteract receptor autophosphorylation and downstream signaling, as well as by serine and serine/threonine kinases that attenuate InsRec/IRS signaling