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While glutathione itself crosses this barrier poorly, the brain can synthesize glutathione from precursor amino acids, and systemic glutathione elevation may indirectly support brain glutathione status through various mechanisms including reduced oxidative stress on blood-brain barrier integrity and improved availability of glutathione precursors

Lower dACC GABA content was associated with greater positive and negative symptom severity, but did not correlate with markers of inflammation, degree of drug treatment or with glutamate

Week five through six: Gradual improvement begins

Prism 9 (GraphPad) was used to determine statistical significance