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This strategy is currently restricted to three disulfide bonds and the synthesis of targets with more than three disulfide bonds will require the development of additional chemistry for chemoselective and regioselective disulfide bond formation

Preclinical studies have shown that ferroptosis-preventing therapies, such as the GSH precursor N-acetylcysteine (NAC), iron-chelating agents like deferoxamine (DFO) and deferiprone (DFP), and specific ferroptosis inhibitors like ferrostatin-1 (Fer-1), can alleviate neurodegeneration and improve motor symptoms in PD animal models ( In this comprehensive review, we aim to summarize the multifaceted roles of ferroptosis in PD pathogenesis, elucidate the links between PD-associated pathogenic proteins and ferroptosis, and provide an overview of current clinical trials and the potential of ferroptosis in the early diagnosis and treatment of PD

With this in mind, toxicity of copper excess in mammalian cells is explained by obstructing the control of the interactome of copper-sulfur containing redoxomes [166,167,168,169]