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In the body, it regulates metabolism in the epiphysis, increases the sensitivity of the hypothalamus to its natural hormonal influences, and normalizes the function of the anterior pituitary, including its regulation of gonadotropins and melatonin

Since PRDX2 has been shown to be glutathionylated by GSH at its cysteine residues to protect against its hyperperoxidation 41 , it is possible that elevated GSH levels in T315I-Bcr-Abl cells might prevent PRDX2 monomer formation, therefore abrogating ROS-induced replication stress

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