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J Surg Oncol 122(3):495-505, 2020

Macrophage secretion products can also recruit other immune cells to the site of tissue injury which results in the establishment of a chronic inflammatory milieu in the PC [48,92]

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In an immunocompetent mouse model of hepatitis B virus (HBV) infection, SFN treatment significantly promoted the repolarization of macrophages towards the antitumoral M1 phenotype, as evidenced by increased expression of Cd86 and iNOS, and inhibited the expression of Arg1