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Journal of Pharmacological Sciences, 91(4), 267-270

CNS) and thereby reduces insulin-dependent glucose uptake via glucose transporter GLUT4 into muscle and subcutaneous fat tissue but increases insulin-independent glucose uptake via GLUT1 into the brain, SNS/HPA axis, and glucose

Contrarily, in all BPC 157-treated rats, from the very beginning, the lesion was markedly smaller (half the size, given the very large lesion in the controls), with less edema, necrosis of muscle fibers, and acute inflammation with foci of abscessation