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10.1016/j.neuropharm.2013.04.024 Neuropharmacology 16 BellantiF.MatteoM.RolloT.De RosarioF.GrecoP.VendemialeG.et al (2013)
Gliotoxin can target integrins to modify their cysteines by a disulfide bond, thus blocking the binding of arginine-glysine-aspartic acid (RGD)-containing extracellular matrix, leading to the inhibition of focal adhesion kinase and the dephosphorylation of p190RhoGAP, which allows for the activation of RhoA and, subsequently, the activation of ROCK, MKK47/MKK7, and JNK and the induction of Bim phosphorylation, which is a proapoptotic factor
This was exacerbated in people who were obese.[ref] Another recent study showed that MAO-A plays a role in mitral valve remodeling in heart disease, adding to the oxidative stress in the cardiovascular system
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