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Ongoing research continues to explore the role of NSP activation by dipeptidyl peptidase 1 in different disease states and the potential clinical benefits of dipeptidyl peptidase 1 inhibition

Accumulation of waste products, specifically lactate, and decreased oxygen concentrations result in alternations in the microenvironment of the IVD hindering the anabolic process of proteoglycan synthesis

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Similarly, in vivo studies and clinical trials have shown evidence for beta-glucans as potent inducers of macrophage, keratinocyte and fibroblast activation, migration and proliferation