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Funding This work was supported by the Minas Gerais State Agency for Research and Development FAPEMIG (BPD-0077722), the National Council for Scientific and Technological Development CNPq, and the Coordination of Superior Level Staff Improvement CAPES (Finance Code 001)

miR-200a mediates protection of thymosin -4 in cardiac microvascular endothelial cells as a novel mechanism under hypoxia-reoxygenation injury

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The metabolism and clearance of both components may be affected by significant liver disease, and patients with severe hepatic impairment should be monitored closely if treatment is deemed necessary