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An interaction between parkin and its substrate -synuclein is a crucial regulatory component of dopaminergic neuron degeneration, loss-of-function mutations in PARK2 usually cause early-onset autosomal recessive PD (Kitada et al., 1998), whereas disease-associated mutations in -synuclein are usually associated with autosomal dominant PD (Polymeropoulos et al., 1997)

Inflammatory cell infiltration follows tissue injury, initially serving protective functions but potentially causing secondary damage if prolonged or excessive

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