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Many anti-angiogenic therapies, such as VEGF inhibitors, suppress tumor growth and metastasis by blocking angiogenesis and repairing abnormal endothelial cells [199, 200]

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While no approved protocol currently exists for this specific combination, the underlying science suggests that multi-pathway approaches targeting amylin, GIP, and GLP-1 receptors simultaneously may offer superior metabolic benefits compared to single-pathway interventions

Latest developments demonstrate that redox-sensitive nanoparticles, such as disulfide bond-containing nanoparticles, can respond to elevated concentrations of glutathione (GSH) in tumors