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Disorders of metabotropic glutamate (mGlu) receptors and neurotrophins have significant negative effects on neuroplasticity, leading to depression, but LAC regulate brain type 2 metabotropic glutamate (mGlu2) receptors and N-methyl-D-aspartic acid (NMDA) receptors to induce antidepressant effect (8385)

doi: 10.1111/j.1523-1755.2005.00273.x

FIN56, an inducer of ferroptosis derived from CIL56, promotes endogenous antioxidant activity by either facilitating the degradation of GPX4 or binding to and activating squalene synthase (SQS), which results in the depletion of CoQ 10 [135]

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